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How to Prevent COPD in Railroad Yards and Locomotive Shops

COPD is a progressive. Symptoms get worse over time and breathing difficulty becomes more difficult.

Research has shown that railroad workers are at a higher risk of developing COPD due to their daily exposure to welding fumes and diesel exhaust. Other risk factors include smoking and alpha-1-antitrypsin deficiencies, a rare genetic disorder that causes emphysema.

Exposure to Diesel Exhaust

Diesel fuel combustion releases a large amount of harmful chemicals into the air. These chemicals are ultrafine particles coated with organic compounds which could cause irritation in the lungs. The particles are so small that they can get into the smallest crevices within your lung. The irritation could lead to lung diseases like COPD (chronic obstruction of the lungs) and asthma.

Research suggests that long-term diesel exhaust fumes exposure may cause COPD. This condition causes breathing difficulties because of the buildup in the lungs. Studies have shown that COPD is linked to occupational exposure to fumes and dust generally, but Railroad Chronic Lymphocytic Leukemia workers are particularly vulnerable.

Diesel exhaust can increase the risk of COPD and other illnesses. A study of railroad employee medical records and job reports discovered that those exposed to the highest levels of diesel emissions - brakemen, train conductors and engineers had an increased risk of lung cancer than other workers.

Freight railroads such as CSX and Norfolk Southern have known for many years that their workers are at risk from diesel fume exposure. In fact, our lawyers have reviewed documents from the Railroad Lung Cancer companies that show that their own medical professionals and claim representatives have held seminars and meetings focused on the issue as early as the 1930's.

Exposure to welding fumes

In locomotive shops and Railroad Pulmonary Fibrosis yards, welding is often required. The process produces fumes that can cause emphysema in people who breathe these fumes regularly. Therefore, it is imperative that welders wear a welding helmet and other respiratory protection. These fumes pose both chronic and acute health hazards. Chronic effects can manifest weeks, months, or even years after exposure.

The fumes from welding can include a variety of chemicals according to the rods as well as the metals being to be welded. Zinc, lead, cadmium and iron are some of the most common elements. In addition the toxins chromium manganese, nickel, and copper have been found in the fumes. Other chemicals, including nitrogen peroxide and oxygen are also found in the fumes. The fumes can cause lung cancer and are believed to be cancerous.

The FELA laws stipulate that a person suffering from an occupational lung disease is able to file a claim against the Railroad Scleroderma for compensation. Railroad workers are susceptible to lung conditions linked to welding fumes, diesel exhaust, and asbestos. Families of people who suffer from COPD (such as emphysema) are encouraged to talk with a Railroad Scleroderma worker FELA attorney for assistance.

Smoking

Combining diesel exhaust with cigarette smoking can increase the risk of COPD. Workers may be exposed the fumes from trains or working in the yard around idling locomotives. Smoking also worsens the symptoms of COPD and can cause them to appear sooner.

A longitudinal cohort study has revealed that railroad workers exposed to diesel exhaust for a prolonged time are at an increased possibility of COPD death. This is no matter if the worker is employed prior or after the introduction of diesel locomotives. After taking into account smoking history, the connection between the duration of diesel exhaust exposure and COPD mortality persists.

COPD is a complicated and debilitating illness that requires medical attention from a physician. There is no cure for COPD but it is crucial to follow the prescribed treatment plan and railroad COPD see your doctor regularly. It is also essential to get pneumonia and flu vaccinations, exercise regularly and do breathing exercises regularly.

Symptoms of COPD include shortness of breath, wheezing and coughing, fatigue and difficulty sleeping. It is sometimes difficult to recognize the condition because many of the signs that are early are similar to other respiratory illnesses, such as a cold or sinus infection. If you or someone you love has been diagnosed with COPD then contact a dedicated railroad COPD attorney from Doran & Murphy to discuss your options.

Genetics

Genetics is the study of how genes or groups of genes affect health and illness. Genes are units of information that determine our physical characteristics, such as hair colour or the likelihood of developing certain diseases, like cancer or heart disease. Genetics research can also help us understand how certain diseases run in families or why we're more likely to develop an illness based on genetic makeup.

By examining lung function measurements of population-based cohorts as well as genetic links with COPD were identified. These studies can reveal genetic variants that are associated with COPD severity or prevalence since COPD is associated with a decrease in lung function. These studies are typically referred to by the term genome-wide associations studies (GWAS).

In several studies, researchers have chosen specific regions or genes to determine their connection with COPD. But candidate genes rarely achieve genome-wide significance and the majority discovered associations have only minor effects sizes.

Recent studies have employed modern genetic technologies in order to better understand COPD pathogenesis. Particularly, techniques using genomics like GWAS and integrative functional genomics have been employed to study the function of specific genes in COPD development and progression. Cho and colleagues 48, for example used GWAS to identify genomic regions associated with COPD severity or prevalence in the COPDGene cohort. This analysis demonstrated that there was a strong correlation between the 15q25 region of the chromosome that contains genes like HHIP CHRNA3/5 and IREB2. The CHRNA3/5 appears to influence COPD risk partly through its influence on tobacco smoking behavior. Variants near the IREB2 genes may confer COPD risk independently of smoking habits, through effects on the metabolic processes of iron.