And catalase contributes to oxidative anxiety and neurogenic hypertension in spontaneously > 자유게시판

And catalase contributes to oxidative worry and neurogenic hypertension in spontaneously hypertensive rats. No cost Radic Biol Med 2006, 40:2028?039. ninety six. Dovinov?I, P ffy R, Gardlik R, Vrankova S, Jendekova L, Kristek F, Sumbalova Z, Paul ova H: Antioxidant gene therapy of hypertension in spontaneously hypertensive rats. In Abstract Book of Satellite symposium on the twenty second Scientific Assembly of Global 2-(2-Aminoethoxy)-5-chloropyridine hydrochloride Culture of Hypertension: 12-13 June 2008. Edited PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/8086425 by Pechanova O. Bratislava: Institute of Ordinary and Pathological Physiology SAS; 2008:forty seven?eight. ninety seven. Carlstrom M, Lai EY, Ma Z, Steege A, Patzak A, Eriksson UJ, Lundberg JO, Wilcox CS, Persson AE: Superoxide dismutase 1 limits renal microvascular reworking and attenuates arteriole and hypertension responses to angiotensin II via modulation of nitric oxide bioavailability. Hypertension 2010, fifty six:907?13. ninety eight. Dikalova AE, Bikineyeva AT, Budzyn K, Nazarewicz RR, McCann L, Lewis W, Harrison DG, Dikalov SI: Therapeutic focusing on of mitochondrial superoxide in hypertension. Circ 2-(2,4-Dichloro-5-fluorophenyl)oxirane Res 2010, 107:106?sixteen.99. Dovinova I, Baranc M, Cac yiov?S, Kristek F, Gajdosechov?L, Gardl R: Modifications in antioxidant reaction and ROS signaling in numerous experimental hypertension styles. J Hypertens 2011, 29(Suppl B):e45. 100. Fukai T, Folz RJ, Landmesser U, Harrison DG: Extracellular superoxide dismutase and cardiovascular disease. Cardiovasc Res 2002, fifty five:239?49. a hundred and one. Gongora MC, Qin Z, Laude K, Kim HW, McCann L, Folz JR, Dikalov S, Fukai T, Harrison DG: Job of extracellular superoxide dismutase in hypertension. Hypertension 2006, forty eight:473?eighty one. 102. Chu Y, Iida S, Lund DD, Weiss RM, DiBona GF, Watanabe Y, Faraci FM, Heistad DD: Gene transfer of extracellular superoxide dismutase lessens arterial tension in spontaneously hypertensive rats: part of heparin-binding area. Circ Res 2003, 92:461?sixty eight.doi:ten.1186/1423-0127-20-69 Cite this informative article as: Majzunova et al.: Redox signaling in pathophysiology of hypertension. Journal of Biomedical Science 2013 twenty:69.Submit your up coming manuscript to BioMed Central and just take full benefit of:?Handy on line submission ?Thorough peer overview ?No room constraints or colour figure PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/25116583 expenses ?Immediate publication on acceptance ?Inclusion in PubMed, CAS, Scopus and Google Scholar ?Investigation that's freely out there for redistributionSubmit your manuscript at www.biomedcentral.com/submit
Mantarro et al. BMC Geriatrics 2012, twelve:69 http://www.biomedcentral.com/1471-2318/12/CASE REPORTOpen AccessAcute portal vein thrombosis precipitated by indomethacin in the HCV-positive aged patientStefania Mantarro1, Marco Tuccori2*, Giuseppe Pasqualetti3, Sara Tognini3, Sabrina Montagnani1, Fabio Monzani3 and Corrado BlandizziAbstractBackground: An elevated possibility of venous thromboembolism has been noted in patients treated with non-steroidal anti-inflammatory medication (NSAIDs). We explain a scenario of acute portal vein thrombosis (PVT) in the hepatitis C virus (HCV)-positive aged affected person following administration of indomethacin. Scenario presentation: A 79-year-old HCV-positive male was hospitalized for severe abdominal soreness, nausea and vomiting, 15 times soon after starting off indomethacin for back suffering. Clinical signs and imaging evaluations disclosed a picture of PVT. Indomethacin was discontinued, plus the patient was begun on fondaparinux and antithrombin. He was discharged 15 days later on resulting from improvement of his medical conditions. Thirty days later on, a follow-up ultrasound didn't display appreciable signals of PVT.